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Jackson Laboratory myd88 flox mice
Myd88 Flox Mice, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/myd88+flox+mice/myd88/pm42302512-55-0-5
Average 86 stars, based on 1 article reviews
myd88 flox mice - by Bioz Stars, 2026-10
86/100 stars

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Related Articles

other:

Article Title: Cancer- and endotoxin-induced cachexia require intact glucocorticoid signaling in skeletal muscle
Article Snippet: Animals Wild-type C57BL/6J mice, MCK-cre, MyD88 Flox, LSL-tdTomato and My88-knockout (My88KO) mice were obtained from The Jackson Laboratories (Bar Harbor, ME, USA).

Article Title: CX 3 CR1 + mononuclear phagocytes support colitis-associated innate lymphoid cell production of IL-22
Article Snippet: C57BL/6, Myd88 flox , CD11c-cre , and Il1r −/− mice were purchased from The Jackson Laboratory.

Article Title: Homeostatic pockets of interferon lambda-stimulated gene production in the intestine are associated with localized exposure to bacterial microbiota
Article Snippet: C57BL/6J (wild-type) mice, Myd88 -/- (B6.129P2(SJL)- Myd88 tm1.1Defr /J, stock #009088), Ticam1 -/- (C57BL/6J- Ticam1 Lps2 /J, stock #005037), Myd88 flox (B6.129P2(SJL)- Myd88 tm1Defr /J, stock #008888), Vav-iCre (B6.Cg- Commd10 Tg(Vav1-icre)A2Kio /J, stock #008610), Vil-cre (B6.Cg-Tg(Vil1-cre)1000Gum/J, stock #021504) mice were originally purchased from Jackson Laboratories and bred in a specific pathogen-free facility at Oregon Health & Science University.

Article Title: IL-1 delineates squalene-based adjuvant efficacy and reactogenicity in a cell-type-specific manner
Article Snippet: C57BL/6JJcl mice (6–8 weeks old) were purchased from CLEA Japan, Inc. (Tokyo, Japan).

Article Title: MyD88 signaling in trophoblasts contributes to maternal high fat diet-associated alterations in placental gene expression and lipid profiles.
Article Snippet: Introduction: Maternal obesity contributes to programming of offspring obesity.. A causal pathway for placental inflammatory changes in utero with obesity remains to be established.. We examined the role of the toll-like receptor (TLR) pathway in mediating maternal obesity associated changes in placental gene expression using trophoblast-specific deletion of the TLR adapter protein, MyD88.

Article Title: Cancer- and endotoxin-induced cachexia require intact glucocorticoid signaling in skeletal muscle
Article Snippet: Wild-type C57BL/6J mice, MCK-cre, MyD88 Flox, LSL-tdTomato and My88-knockout (My88KO) mice were obtained from The Jackson Laboratories (Bar Harbor, ME, USA).

Knock-Out:

Article Title: TLR4 Activation Enhances the PD-L1-Mediated Tolerogenic Capacity of Colonic CD90 + Stromal Cells
Article Snippet: .. MyD88 flox mice were obtained from Jackson laboratory and were crossed with fibroblast specific Cre mice (TgCol1α2-CreERTM) in order to generate fibroblast specific Tamoxifen-inducible conditional knockout mice under fibroblast specific Col1α2 promoter selective for MyD88 (Myd88 flox TgCol1α2-CreERTM or Fib-Myd88 KO). ..

Transgenic Assay:

Article Title: TLR signaling adaptor protein MyD88 in primary sensory neurons contributes to persistent inflammatory and neuropathic pain and neuroinflammation
Article Snippet: .. MyD88 flox mice were purchased from Jackson Labs. Na v 1.8 cre transgenic mice were kindly provided by Rohini Kuner (University of Heidelberg) . ..



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